Polyenylpyrrole Derivatives Inhibit NLRP3 Inflammasome Activation and Inflammatory Mediator Expression by Reducing Reactive Oxygen Species Production and Mitogen-Activated Protein Kinase Activation | PLOS ONE
Punicalin ameliorates cell pyroptosis induced by LPS/ATP thr | JIR
NLRP1 and NLRP3 inflammasomes mediate LPS/ATP‑induced pyroptosis in knee osteoarthritis
Molecular hydrogen inhibits lipopolysaccharide-triggered NLRP3 inflammasome activation in macrophages by targeting the mitochondrial reactive oxygen species - ScienceDirect
Autocrine Regulation of Macrophage Activation via Exocytosis of ATP and Activation of P2Y11 Receptor | PLOS ONE
Chronic TLR Stimulation Controls NLRP3 Inflammasome Activation through IL-10 Mediated Regulation of NLRP3 Expression and Caspase-8 Activation | Scientific Reports
Mechanisms that lead to the regulation of NLRP3 inflammasome expression and activation in human dental pulp fibroblasts - ScienceDirect
Exogenous hydrogen sulfide mitigates LPS + ATP-induced inflammation by inhibiting NLRP3 inflammasome activation and promoting autophagy in L02 cells | SpringerLink
ATP/P2X7 receptor signaling as a potential anti-inflammatory target of natural polyphenols | PLOS ONE
Molecules | Free Full-Text | Juglone Suppresses LPS-induced Inflammatory Responses and NLRP3 Activation in Macrophages
Effect of LPS and ATP on the pyroptosis of J774 cells | Download Scientific Diagram
Icariside I specifically facilitates ATP or nigericin-induced NLRP3 inflammasome activation and causes idiosyncratic hepatotoxicity | Cell Communication and Signaling | Full Text
Lipopolysaccharide/adenosine triphosphate induces IL‑1β and IL-18 secretion through the NLRP3 inflammasome in RAW264.7 murine macrophage cells
Chronic TLR Stimulation Controls NLRP3 Inflammasome Activation through IL-10 Mediated Regulation of NLRP3 Expression and Caspase-8 Activation | Scientific Reports
Lipopolysaccharide/adenosine triphosphate induces IL‑1β and IL-18 secretion through the NLRP3 inflammasome in RAW264.7 murine macrophage cells
Proposed anti-inflammatory mechanism of compound 1h in LPS+ATP-activated macrophages.
Neutrophil P2X7 receptors mediate NLRP3 inflammasome-dependent IL-1β secretion in response to ATP | Nature Communications
ATP is released by monocytes stimulated with pathogen-sensing receptor ligands and induces IL-1β and IL-18 secretion in an autocrine way | PNAS
Frontiers | Interplay Between NLRP3 Inflammasome and Autophagy
Anti-NLRP3 Inflammasome Natural Compounds | Encyclopedia MDPI
Lipopolysaccharide/adenosine triphosphate induces IL‑1β and IL-18 secretion through the NLRP3 inflammasome in RAW264.7 murine macrophage cells
Caffeine Inhibits NLRP3 Inflammasome Activation by Suppressing MAPK/NF-κB and A2aR Signaling in LPS-Induced THP-1 Macrophages
BMDC secrete mature IL-1β following LPS priming and ATP. Day 8 BMDC at... | Download Scientific Diagram
